Present study was designed to find out the relationship between emotional intelligence and self esteem
among sighted and visually impaired school students. The main objectives of this study were to measure self
esteem of sighted and visually impaired students, to measure emotional intelligence level of sighted and
visually impaired students, to compare self esteem and emotional intelligence of sighted and visually impaired
students, to compare emotional intelligence and self esteem of sighted and visually impaired students
studying in private and public sectors institutions and to investigate the role of students' demographic
variations of gender, age, grade, family socioeconomic status, number of siblings, birth order, parental
professions and qualification in determining their self esteem and emotional intelligence levels.
In this study standardized questionnaires were used to measure emotional intelligence and self esteem,
emotional intelligence was measured by using Emotional Intelligence Scale (EIS) and Self Esteem
Questionnaire (SEQ) was used to measure students' self esteem. EIS contained 33 items comprised seven
subscales i.e. Appraisal of Emotions in Self, Appraisal of Emotions in Others, Emotional Expression, Emotional
Regulation of others, Utilization of Emotions in Problem Solving and Uncategorized. SEQ contained 36 items
comprised five subscales i.e. self — Regard, Social Confidence, School Abilities, Physical Appearance and
Physical Abilities. Cronbach's Alpha reliability of EIS and SEQ were .843 and .696. A sample comprising of 200
students was taken from sighted and visually impaired students studying at secondary schools from
Islamabad, Rawalpindi, Sargodha District and Attock. Stratified random sampling technique was used to
collect data for the study.
Research findings revealed that there is significant difference in responses between sighted and visually
impaired school students regarding emotional intelligence and self esteem, this study also reveals that there
is significant positive relationship between emotional intelligence and self esteem.
دن بھی دیکھا ، رات بھی دیکھی لہجے دیکھے ، بات بھی دیکھی شام کو ہم نے یاد بھی دیکھی جیت بھی دیکھی ، مات بھی دیکھی دن کو ہم نے تارے دیکھے لہجے تیرے سارے دیکھے
ہجر بھی دیکھا ، وصل بھی دیکھا چاند بھی دیکھا ، بادل دیکھے
کیسے کیسے وہم تھے دل میں ہنستے ہنستے رونے لگتے روتے روتے ہنسنے لگتے ایسے تُو نے پاگل دیکھے
عمر گزاری ہے تجھ بن میں آج بھی زندہ ہوں تجھ بن میں لیکن اپنا حال تو دیکھو دن سویا سویا رہتا ہے شب جاگی جاگی رہتی ہے
Our research paper is about to know that how Islamic work ethic (IWE) on organizational citizenship behaviors (OCBs), Knowledge sharing behavior (KSBs) and organizational commitment between workers of colleges of Hajeera, district Rawalakot, Azad Jammu Kashmir. We take 210 workers of different colleges from Azad Jammu Kashmir for the purpose of this study. Result indicates that Islamic work Ethics (IWE) plays a strong impact on Organizational Citizenship behavior (OCB). In short one with having high Islamic work Ethics (IWE) possess more organizational citizenship behavior (OCB) as one who possess low Islamic work Ethics (IWE). Our results are indicating (IWE) has a direct relationship with Knowledge sharing behavior (KSB). One with high Islamic work Ethics (IWE) possess high Knowledge sharing behavior (KSB) as one with having low Islamic work Ethics (IWE) possess low Knowledge sharing behavior (KSB). In this paper we will show you theoretical & practical implications of our results.
Introduction: The World Health Organization defines infertility as the failure of achieving conception after one year of unprotected intercourse. Worldwide, approximately 15% of couples are affected by infertility and genetic anomalies account for 15-30% of male factor infertility. Nearly 15% of infertile males suffer from azoospermia in the form of obstructive azoospermia or non-obstructive spermatogenic failure. Polycystic Ovary Syndrome (PCOS) was contributing 6-10% infertility in female population. Environmental and genetic factors are involved. However; etiology of PCOS still remains debatable. Primary Ovarian Insufficiency (POI) or Premature Ovarian Failure (POF) affects 1- 2% of women, and is characterized by amenorrhea before the age of 40 years. POI is heritable in up to 30% of individuals. Methodology: The study presented in the dissertation describes clinical and genetic analysis of twelve Pakistani infertile families (A-L) exhibiting azoospermia, POF, PCOS, Y chromosome microdeletions and chromosomal aberrations. These families were collected from different districts of Khyber Pakhtunkhwa Pakistan. Informed consents were taken from all the participants. Results: In this study, we investigated four azoospermic families (A-D) by whole exome sequencing (WES) analysis. WES data analysis of family A with two males with obstructive azoospermia and two fertile members (mother and brother) revealed a novel nonsense variant c.2326C>T (p.R776X) in dominant X-linked ADGRG2. WES data analysis and Sanger sequencing of family B with one azoospermic male, one fertile brother and parents revealed a compound heterozygous variant in AFF4, including c.3319A>G (p.T1107A). In family C one proband and parents samples were subjected for WES analysis, but no pathogenic variants were identified. WES data analysis of family D with one infertile brother, one fertile brother and parents revealed a novel nonsense variant c.646G>A (p.G216R) in X-linked AR. In family E, WES was performed for four family members and seven potential variants were identified but Sanger sequencing failed to confirm any pathogenic variant. Family F has two daughters displaying primary amenorrhea, elevated LH/FSH levels, atrophic uteri, reduced ovarian reserves, and normal 46XX karyotypes. WES analysis of five family members (parents, two affected daughters and one unaffected daughter) revealed a novel frame shift variant (c.709delC, p.Leu237fs) in the luteinizing hormone/choriogonadotropin receptor (LHCGR) gene. PCOS Families G, H and I were investigated for pathogenic variants. SNP microarray and WES analysis failed to identify pathogenic variants in PCOS families. Family J showed Y chromosome microdeletions (AZFc) in two azoospermic brothers. Families K and L exhibited chromosomal aberrations; OX and XXY karyotypes in azoospermic members in families respectively. Conclusion: Four families revealed novel variants as the likely cause of infertility (ADGRG2, nonsense variant (c.2326C>T); AR nonsense variant (c.646G>A); AFF4 compound heterozygous (c.3319A>G); LHCGR, frame-shift variant (c.709delC). WES in four families (three PCOS one POF) families could not identify genetic causes in the coding region. One family exhibited “Y” chromosome microdeletion (AZFc region deletion) and two families were found with chromosomal aberrations (XXY and OX).